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Dengue fever is a flu-like viral disease common over the tropical and sub-tropical locations around the world, primarily in city and peri-urban areas. Today, it affects an estimated 50 million to 100 mil in the tropics (Epstein, 2000). The disease has 4 antigenically related serotypes, which can be named DEN-1, DEN-2, DEN-3, and DEN-4. Each melindre serotype is a variation of the flavivrus genus. Dengue can be spread by aedes aegypti, a domestic, day-biting bug that prefers to bite human beings. Currently, there is no vaccine offered to prevent melindre.

Each type with the dengue virus is re-emerging worldwide, especially in the Western Hemisphere. Research has demonstrated that a lot of factors will be contributing to the resurgence of dengue fever such as uncontrolled urbanization, improved international travel, substandard socio-economical conditions, and ultimately global warming. Around the world has shown to become major contributor to the spread of dengue fever.

Over a molecular level, dengue fever is labeled as a flavivirus and shows up as a spherical particle, regarding 40 to 50 nanometers in size (www.malarde.pf...). We have a lipid envelope enclosing a nucleocapsid key (www.malarde.pf/...). The dengue disease genome is a single-strand confident sense RNA of approximately 11 kilobases (www.malarde.pf/...). The fully developed virions include three strength proteins: C (capsid), prM (membrane) and E (envelope) and six nonstructural protein (www.malarde.pf/...).

When a person is usually infected simply by dengue fever, symptoms will start to appear from four to six times after the pathogen has been transmitted from the afflicted mosquito for the person (http://astdhpphe.org...). The malware can not be distributed from an infected person to another person. However , in the event that another mosquito bites the infected person, the virus can be sent to another person through that newly afflicted mosquito. The infected person remains a viral supply for additional mosquitoes for approximately 6 days...

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